Fig. 6
From: Status of kinases in Epstein-Barr virus and Helicobacter pylori Coinfection in gastric Cancer cells
Graphical abstract of the experimental outcome in the report. H. pylori secretory molecule activates two kinases FYN and ITK, which may activate the effector molecule leading to the nucleus for cell proliferation, morphological changes, and cytoskeleton rearrangement. H. pylori adherence activates TYK2 kinase and inhibits apoptotic genes like FASL, FASR, and APAF-1, which contribute to cell proliferation