From: Pancreatic tumor in type 1 autoimmune pancreatitis: a diagnostic challenge
 | AIP (n = 19) | AIP + PaC (n = 1) | P Value |
---|---|---|---|
Pancreatic parenchyma | |||
 Enlargement pattern |  |  | 0.250 |
  Diffuse | 15 (79%) | 0 (0%) |  |
  Focal | 4 (21%) | 1 (100%) |  |
 Presence of atrophy | 2 (11%) | 1 (100%) | 0.150 |
 Signal intensity on T1WI | |||
  Hypointense | 16 (84%) | 1 (100%) | 0.911 |
  Isointense | 2 (11%) | 0 (0%) |  |
  Hyperintense | 1 (5%) | 0 (0%) |  |
  Presence of heterogeneity | 5 (26%) | 0 (0%) | 1.000 |
 Signal intensity on T2WI | |||
  Hypointense | 0 (0%) | 0 (0%) | 0.452 |
  Isointense | 7 (37%) | 0 (0%) |  |
  Hyperintense | 12 (63%) | 1 (100%) |  |
  Presence of heterogeneity | 6 (31%) | 1 (100%) | 0.300 |
 Signal intensity on pancreatic phase | |||
  Hypointense | 13 (69%) | 1 (100%) | 0.502 |
  Isointense | 6 (31%) | 0 (0%) |  |
  Hyperintense | 0 (0%) | 0 (0%) |  |
  Presence of heterogeneity | 12 (63%) | 1 (100%) | 1.000 |
 Signal intensity on portal venous phase | |||
  Hypointense | 1 (5%) | 0 (0%) | 0.798 |
  Isointense | 5 (26%) | 0 (100%) |  |
  Hyperintense | 13 (69%) | 1 (0%) |  |
  Presence of heterogeneity | 6 (31%) | 1 (100%) | 0.350 |
 Signal intensity on delayed venous phase | |||
  Hypointense | 3 (16%) | 0 (0%) | 0.753 |
  Isointense | 12 (63%) | 1 (100%) |  |
  Hyperintense | 4 (21%) | 0 (0%) |  |
  Presence of heterogeneity | 5 (26%) | 1 (100%) | 0.300 |
 Presence of rim enhancement | 0 (0%) | 1 (100%) | 0.050 |
Main pancreatic duct | |||
 Presence of dilatation | 3 (16%) | 0 (0%) | 1.000 |
 Presence of cutoff | 6 (31%) | 1 (100%) | 0.350 |
Peripancreatic structures | |||
 Presence of halo sign | 1 (5%) | 0 (0%) | 1.000 |
 Presence of stranding | 0 (0%) | 0 (0%) | 1.000 |
 Presence of ascites | 0 (0%) | 0 (0%) | 1.000 |
 Presence of vessel stricture | 13 (69%) | 1 (100%) | 1.000 |