Fig. 7From: Leptin-induced ER-α-positive breast cancer cell viability and migration is mediated by suppressing CCN5-signaling via activating JAK/AKT/STAT-pathwayA Model depicting the role of CCN5 on leptin-induced cell viability, EMT, cell migration and Stemness. Leptin blocks CCN5 expression via activating JAK/ AKT /STAT3-signaling in luminal (ER-positive) BC cells to promote cell viability and aggressive phenotypes of these cellsBack to article page