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Fig. 5 | BMC Cancer

Fig. 5

From: Increased toll-like receptors and p53 levels regulate apoptosis and angiogenesis in non-muscle invasive bladder cancer: mechanism of action of P-MAPA biological response modifier

Fig. 5

Immunolabelled antigen intensities of the urinary bladder from the CONTROL (a, b, c, d, e, f), MNU (g, h, i, j, k, l), MNU-BCG (m, n, o, p, q, r), and MNU-P-MAPA (s, t, u, v, w, x) groups. TLR4 immunoreactivities (asterisks) were moderate in the urothelium from the CONTROL group (a), weak in the MNU group (g) and intense in the MNU-BCG (m) and MNU-P-MAPA (s) groups. TRIF immunoreactivities (asterisks) were weak in the urothelium from the CONTROL (b) and MNU (h) groups, moderate in the MNU-BCG (n) group and intense in the MNU-P-MAPA (t) group. IRF-3 immunoreactivities (arrows) were weak in the urothelium from the CONTROL (c) and MNU (i) groups, moderate in the MNU-BCG (o) group and intense in the MNU-P-MAPA (u) group. IFN-γ immunoreactivities (arrows) were weak in the urothelium from the CONTROL (d) and MNU (j) groups, moderate in the MNU-BCG (p) group and intense in the MNU-P-MAPA (v) group. iNOS immunoreactivities (asterisks) were weak in the urothelium from the CONTROL (e) and MNU (k) groups, moderate in the MNU-BCG (q) group and intense in the MNU-P-MAPA (w) group. BAX immunoreactivities (asterisks) were weak in the urothelium from the CONTROL (f) group, moderate in the MNU (l) and MNU-BCG (r) groups and intense in the MNU-P-MAPA (x) group. a–x Ur urothelium

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BMC Cancer

ISSN: 1471-2407

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