Figure 3
From: PPARγ-independent induction of growth arrest and apoptosis in prostate and bladder carcinoma
Effect of PPARγ ligands on cell number of TSU-Pr1/TSU-Pr1-B1/TSU-Pr1-B2 series day 5. TSU-Pr1, TSU-Pr1-B1 and TSU-Pr1-B2 cells were treated with control (0.2% DMSO), GW9662 (10 μM), troglitazone (10 and 100 μM; TGZ), ciglitazone (40 μM; CGZ), rosiglitazone (10 μM; RGZ) and 15-deoxy-prostaglandin J2 (1 and 10 μM; 15dPGJ2) in the presence or absence of antagonist GW9662 (10 μM) at day 0. Values are mean ± SEM from Day 5. * All cell lines significantly different to the respective controls, # TSU-Pr1 and TSU-Pr1-B2 significantly different to the respective controls (unpaired t test).