Figure 6
From: Decreased miR-106a inhibits glioma cell glucose uptake and proliferation by targeting SLC2A3 in GBM
Expression of SLC2A3 abrogates miR-106a-mediated glycolysis and proliferation. (A) Cells were transfected with SLC2A3 plasmid (without the 3′UTR) and miR-106a. SLC2A3 protein level was detected by Western blot assay. GAPDH protein was regarded as endogenous normalizer. (B-D) Cells were transfected with SLC2A3 plasmid (without the 3′UTR) and miR-106a. Cell cycle, glucose uptake and intracellular ATP production assays were performed respectively. Data were expressed as mean ± SD of 3 independent experiments.