Fig. 6From: Integration of clinical and transcriptomics reveals programming of the lipid metabolism in gastric cancerACLY increased the expression level of fatty acid synthesis enzymes and AKT/mTOR signaling. Intracellular levels of free fatty acid (A) and triglyceride (B) with ACLY knocked-down or over-expressed; Quantitative RT-PCR analysis for mRNA levels of lipogenic enzymes with ACLY knocked-down (C) and over-expression (D); E Spearman correlation analysis of the mRNA expression levels of ACLY and lipogenic enzymes; Colony formation assay (F) and Transwell assay (G) in GC cells with ACLY knocked-down or over-expressed; BGC823 (H) and SGC7901 (I) expressing ectopic ACLY or vector were analyzed for mTOR signaling by immunoblottingBack to article page