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Fig. 8 | BMC Cancer

Fig. 8

From: FAM168A participates in the development of chronic myeloid leukemia via BCR-ABL1/AKT1/NFκB pathway

Fig. 8

Clinical specimens verify the role of FAM168A in CML. PBMCs were isolated from the peripheral blood of 3 healthy controls (NC1, NC2, NC3) and 3 CML patients (CML1, CML2, and CML3). Proteins were extracted and FAM168A, AKT1, p-AKT1, IκB-α, and Cyclin B1were detected by Western blot. a The Western blot images of FAM168A, AKT1, p-AKT1, IκB-α and Cyclin B1. b the average expression of FAM168A, AKT1, p-AKT1, IκB-α and Cyclin B1. Data were presented as mean ± SD from three independent experiments. **p < 0.01

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