Fig. 1
From: FAM168A participates in the development of chronic myeloid leukemia via BCR-ABL1/AKT1/NFκB pathway
FAM168A expression in PBMCs and K562 cells. a PBMCs were isolated from the peripheral blood of three healthy children and three CML patients. The FAM168A mRNA expression was higher in CML patients and K562 cells. b The mean FAM168A mRNA expression was significantly higher in CML patients and K562 cells competed to healthy controls. c Western blot images of FAM168A protein expression in PBMCs from CML patients, healthy controls and K562 cells. d The mean FAM168A protein expression in CML patients, healthy controls and K562 cells. Data were presented as mean ± SD from three independent experiments. **p < 0.01