Fig. 3From: PPARγ activation serves as therapeutic strategy against bladder cancer via inhibiting PI3K-Akt signaling pathwayPPARγ agonists induced cell cycle G2 arrest in bladder cancer cells. (a) Cell cycle determined with flow cytometry in Umuc-3 cells. (b) Cell cycle determined with flow cytometry in 5637 cells. (c) Cell cycle distribution Umuc-3 cells. (d) Cell cycle distribution in 5637 cells. Umuc-3 and 5637 cells were treated with pioglitazone, rosiglitazone, T0070907, and GW9662 (10, 20 μM) for 72 hBack to article page