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Fig. 3 | BMC Cancer

Fig. 3

From: Increased toll-like receptors and p53 levels regulate apoptosis and angiogenesis in non-muscle invasive bladder cancer: mechanism of action of P-MAPA biological response modifier

Fig. 3

Immunolabelled antigen intensities of the urinary bladder from the CONTROL (a, b, c, d, e, f), MNU (g, h, i, j, k, l), MNU-BCG (m, n, o, p, q, r), and MNU-P-MAPA (s, t, u, v, w, x) groups. TLR2 immunoreactivities (asterisks) were moderate in the urothelium from the CONTROL (a) group, weak in the MNU (g) group and intense in the MNU-BCG (m) and MNU-P-MAPA (s) groups. MyD88 immunoreactivities (asterisks) were moderate in the urothelium from the CONTROL (b) group, weak in the MNU (h) group and intense in the MNU-BCG (n) and MNU-P-MAPA (t) groups. IKK-α immunoreactivities (arrows) were weak in the urothelium from the CONTROL (c) group, moderate in the MNU (i) group, intense in the MNU-BCG group (o) and weak in the MNU-P-MAPA (u) group. NF-kB immunoreactivities (arrows) were weak in the cytoplasm of the urothelial cells from the CONTROL (d) group, intense in the nucleus and cytoplasm of the urothelial cells from the MNU (j) group, moderate in the nucleus and cytoplasm of the urothelial cells from the MNU-BCG (p) group and weak in the cytoplasm of the urothelial cells from the MNU-P-MAPA (v) group. TNF-α immunoreactivities (asterisks) were weak in the urothelium from the CONTROL (e) group, intense in the MNU (k) and MNU-BCG (q) groups and weak in the MNU-P-MAPA (w) group. IL-6 immunoreactivities (asterisks) were weak in the urothelium from the CONTROL (f) group, intense in the MNU (l) and MNU-BCG (r) groups and weak in the MNU-P-MAPA (x) group. a–x Ur urothelium

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BMC Cancer

ISSN: 1471-2407

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