Cell lines
|
CDKN2Adeletion
|
BAP1
|
NF2
|
TP53
|
EGFR
|
KRAS
|
NRAS
|
BRAF
|
PIK3 CA
|
ERBB2
|
AKT1
|
---|
NCI-Meso16
|
wt
|
Intron 4 spice site mutation c.256-2A > G, Hm
|
Wt
|
Wt
|
Wt
|
Wt
|
Wt
|
Wt
|
Wt
|
Wt
|
Wt
|
NCI-Meso17
|
Hm
|
Deletion of CAGAT at intron 15/exon 16 junction. c.
|
Wt
|
Wt
|
Wt
|
Wt
|
Wt
|
Wt
|
Wt
|
Wt
|
Wt
|
1984-3_1985delCAGAT
|
NCI-Meso18
|
Hm
|
Large deletion
|
Wt
|
Wt
|
Wt
|
Wt
|
Wt
|
Wt
|
Wt
|
Wt
|
Wt
|
NCI-Meso19
|
Hm
|
Wt
|
Large deletion
|
*P322S
|
Wt
|
Wt
|
Wt
|
Wt
|
Wt
|
Wt
|
Wt
|
NCI-Meso21
|
Hz
|
Homozygous 20 bp deletion in exon 13. c.
|
Wt
|
Wt
|
Wt
|
Wt
|
Wt
|
Wt
|
Wt
|
Wt
|
Wt
|
1302_1321delTGGGCAA CTGTCAGTGCTGC
|
- All 5 cell lines were analyzed for mutations in BAP1 whole gene, P53 exons 2–9, EGFR Exons 18–24, KRAS and NRAS Codon 12, 13 and 61, BRAF-599-601, PIK3CA Exon 9 and 20, ERBB2 insertion in Exon 20 and AKT1 codon 17. *TP53 heterozygous mis-sense mutation in codon 322 (P322S, Exon 9), polymorphism codon 72 (P72R, Exon 4). Hm, homozygous. Hz, heterozygous. Wt, wild-type.