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Table 3  hOGG1  Ser326Cys genotype frequency and distribution according to smoking status

From: An association between hOGG1Ser326Cys polymorphism and the risk of bladder cancer in non-smokers: a meta-analysis

  First author [reference] Genotype
   Cases n (%) Controls n (%)
   Ser/Ser Ser/Cys Cys/Cys Ser/Ser Ser/Cys Cys/Cys
Smoker Karahalil (2006)[23] 14 (41.2%) 16 (47.1%) 4 (11.7%) 27 (62.8%) 9 (20.9%) 7 (16.3%)
  Huang (2007) [21] 279 (62.7%) 166 a (37.3%) 183 (55.5%) 147a (44.5%)
  Arizono (2008) [17] 42 (25.5%) 72 (43.6%) 51 (30.9%) 42 (26.6%) 83 (52.5%) 33 (20.9%)
  Mittal (2011) [31] 107 b (89.2%) 13 (10.8%) 72 b (93.5%) 5 (6.49%)
Non-smoker Karahalil (2006) [23] 7 (53.8%) 4 (30.8%) 2 (15.4%) 38 (66.7%) 9 (15.8%) 10 (17.5%)
  Huang (2007) [21] 96 (60.8%) 62 b (39.2%) 165 (59.4%) 113 b (40.6%)
  Arizono (2008) [17] 19 (22.1%) 35 (40.7%) 32 (37.2%) 25 (26.9%) 52 (55.9%) 16 (17.2%)
  Mittal (2011) [31] 75 a (84.3%) 14 (15.7%) 161 a (93.1%) 12 (6.94%)
  1. a: As only the data of dominant model were available, these data were referred to the sum of Ser/Cys and Cys/Cys.
  2. b: As only the data of recessive model were available, these data were referred to the sum of Ser/ Ser and Ser/Cys.