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Table 3  hOGG1  Ser326Cys genotype frequency and distribution according to smoking status

From: An association between hOGG1Ser326Cys polymorphism and the risk of bladder cancer in non-smokers: a meta-analysis

 

First author [reference]

Genotype

  

Cases n (%)

Controls n (%)

  

Ser/Ser

Ser/Cys

Cys/Cys

Ser/Ser

Ser/Cys

Cys/Cys

Smoker

Karahalil (2006)[23]

14 (41.2%)

16 (47.1%)

4 (11.7%)

27 (62.8%)

9 (20.9%)

7 (16.3%)

 

Huang (2007) [21]

279 (62.7%)

166 a (37.3%)

183 (55.5%)

147a (44.5%)

 

Arizono (2008) [17]

42 (25.5%)

72 (43.6%)

51 (30.9%)

42 (26.6%)

83 (52.5%)

33 (20.9%)

 

Mittal (2011) [31]

107 b (89.2%)

13 (10.8%)

72 b (93.5%)

5 (6.49%)

Non-smoker

Karahalil (2006) [23]

7 (53.8%)

4 (30.8%)

2 (15.4%)

38 (66.7%)

9 (15.8%)

10 (17.5%)

 

Huang (2007) [21]

96 (60.8%)

62 b (39.2%)

165 (59.4%)

113 b (40.6%)

 

Arizono (2008) [17]

19 (22.1%)

35 (40.7%)

32 (37.2%)

25 (26.9%)

52 (55.9%)

16 (17.2%)

 

Mittal (2011) [31]

75 a (84.3%)

14 (15.7%)

161 a (93.1%)

12 (6.94%)

  1. a: As only the data of dominant model were available, these data were referred to the sum of Ser/Cys and Cys/Cys.
  2. b: As only the data of recessive model were available, these data were referred to the sum of Ser/ Ser and Ser/Cys.